منابع مشابه
Antidepressants and brain respiration.
Imipramine and clorgyline, at concentrations of 0.002 M, inhibit the respiration of brain tissue by 82 and 71 per cent respectively, while chloropromazine and tranylcypromine, at concentrations of 0.01 M, inhibit it about 25 per cent. Deprenyl and amphetamine at a concentration of 0.002 M inhibit brain tissue respiration by 12 and 18 per cent respectively. Respiration in brain is least affected...
متن کاملThe Respiration of the Developing Brain
This report deals with the oxygen consumption of different parts of the brains of rats during extrauterine development and the glucose utjilization of whole brains at various ages. METHODS. The brain, without the olfactory bulbs, was divided as follows: The pallium and cerebellum were separated from the neuraxis. The latter was cut in two in front of the pons. The caudal part to the first spina...
متن کاملThe in vitro respiration of normal brain and brain tumors.
The respiratory rates of normal and neoplastic cerebral tissues, and the effect of various anticancer drugs upon them, were measured according to the standard in vitro Warburg technic. The temporal and parietal lobes of normal brain had higher respiratory rates than did the occipital and frontal lobes, and gray matter had a much higher respiratory rate than white matter. Primary and metastatic ...
متن کاملEffect of ethanol on respiration of rat-brain-cortex slices.
Anson, M. L. (1938). J. gen. Physiol. 22, 79. Archibald, W. J. (1947). J. phys. Chem. 51, 1204. Berridge, N. J. (1955). In Methods in Enzymology, vol. 2, p. 69. Ed. by Colowick, S. P. & Kaplan, N. 0. New York: Academic Press Inc. Blackburn, S. & Lowther, A. G. (1951). Biochem. J. 48, 126. Charlwood, P. A. (1957). Trans. Faraday Soc. 53, 871. Herriott, R. M. (1938a). J. gen. Physiol. 21, 501. He...
متن کاملCannabinoid-induced changes in respiration of brain mitochondria.
Cannabinoids exert various biological effects that are either receptor-mediated or independent of receptor signaling. Mitochondrial effects of cannabinoids were interpreted either as non-receptor-mediated alteration of mitochondrial membranes, or as indirect consequences of activation of plasma membrane type 1 cannabinoid receptors (CB1). Recently, CB1 receptors were confirmed to be localized t...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Biochemical Journal
سال: 1936
ISSN: 0306-3283
DOI: 10.1042/bj0301577